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pre coated microplate strips  (Sino Biological)


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    Structured Review

    Sino Biological pre coated microplate strips
    Pre Coated Microplate Strips, supplied by Sino Biological, used in various techniques. Bioz Stars score: 94/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/pre+coated+microplate+strips/pmc13120400-60-6-9?v=Sino+Biological
    Average 94 stars, based on 2 article reviews
    pre coated microplate strips - by Bioz Stars, 2026-08
    94/100 stars

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    Absolute Biotech Inc pre-coated gal3 specific 96 well strip microplates elisa kit
    Comparison of circulating <t>gal3</t> levels in porcine model (SCD vs. survivors). Panel ( A ) shows comparison of circulating galectin-3 levels in survivors (N = 9) vs. SCD group (N = 5) approximately 3 months after the coronary intervention. Panel ( B ) shows the time-course of circulating galectin-3 increase with higher levels detected ~10 days before ventricular tachycardia/ventricular fibrillation (VT/VF) cardiac arrest, compared to the mean galectin-3 levels measured in the pigs that had no cardiac arrest. N = 3–11, *, p = 0.0036 10 days before cardiac arrest vs. 20 days before cardiac arrest in SCD pigs, †, p = 0.0002 10 days before cardiac arrest in SCD vs. survivors pigs. ( C , D ) shows representative immunofluorescence images of galectin-3-stained pig left ventricular sections with non-SCD and SCD. The bar diagram ( E ) in the lower panel shows a significantly increased level of galectin-3 in these animals. N = 5 per group. CTF, corrected total fluorescent intensity. Scale bar: 50 µm, magnification ×400.
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    Comparison of circulating <t>gal3</t> levels in porcine model (SCD vs. survivors). Panel ( A ) shows comparison of circulating galectin-3 levels in survivors (N = 9) vs. SCD group (N = 5) approximately 3 months after the coronary intervention. Panel ( B ) shows the time-course of circulating galectin-3 increase with higher levels detected ~10 days before ventricular tachycardia/ventricular fibrillation (VT/VF) cardiac arrest, compared to the mean galectin-3 levels measured in the pigs that had no cardiac arrest. N = 3–11, *, p = 0.0036 10 days before cardiac arrest vs. 20 days before cardiac arrest in SCD pigs, †, p = 0.0002 10 days before cardiac arrest in SCD vs. survivors pigs. ( C , D ) shows representative immunofluorescence images of galectin-3-stained pig left ventricular sections with non-SCD and SCD. The bar diagram ( E ) in the lower panel shows a significantly increased level of galectin-3 in these animals. N = 5 per group. CTF, corrected total fluorescent intensity. Scale bar: 50 µm, magnification ×400.
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    R&D Systems a pre-coated galectin-3 specific 96-well strip microplate enzyme-linked immunosorbent assays (elisa) kit
    Myocardial gene expression of <t>galectin-3</t> after acute MI in mice. Panel A: Representative Trichrome-labeled section from infarct region. Panel B: Similarly stained section from remote region. The data columns represent fold-change of mRNA expression at different time-points of genomic profiling after acute MI induction. Panel C: Maximal galectin-3 expression was noted at 7-21 days with a chronic elevation present till 90 days post-MI. N = 3, each group; total, 8 time-points. * P < .05, 1-4 days vs 7-21 days; ** P < .05, 7-21 days vs 45-90 days. Panel D: The remote region showed minimal and statistically non-significant galectin-3 mRNA expression over time. MI indicates myocardial infarction.
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    Image Search Results


    Comparison of circulating gal3 levels in porcine model (SCD vs. survivors). Panel ( A ) shows comparison of circulating galectin-3 levels in survivors (N = 9) vs. SCD group (N = 5) approximately 3 months after the coronary intervention. Panel ( B ) shows the time-course of circulating galectin-3 increase with higher levels detected ~10 days before ventricular tachycardia/ventricular fibrillation (VT/VF) cardiac arrest, compared to the mean galectin-3 levels measured in the pigs that had no cardiac arrest. N = 3–11, *, p = 0.0036 10 days before cardiac arrest vs. 20 days before cardiac arrest in SCD pigs, †, p = 0.0002 10 days before cardiac arrest in SCD vs. survivors pigs. ( C , D ) shows representative immunofluorescence images of galectin-3-stained pig left ventricular sections with non-SCD and SCD. The bar diagram ( E ) in the lower panel shows a significantly increased level of galectin-3 in these animals. N = 5 per group. CTF, corrected total fluorescent intensity. Scale bar: 50 µm, magnification ×400.

    Journal: Cells

    Article Title: Galectin-3 Is Associated with Cardiac Fibrosis and an Increased Risk of Sudden Death

    doi: 10.3390/cells12091218

    Figure Lengend Snippet: Comparison of circulating gal3 levels in porcine model (SCD vs. survivors). Panel ( A ) shows comparison of circulating galectin-3 levels in survivors (N = 9) vs. SCD group (N = 5) approximately 3 months after the coronary intervention. Panel ( B ) shows the time-course of circulating galectin-3 increase with higher levels detected ~10 days before ventricular tachycardia/ventricular fibrillation (VT/VF) cardiac arrest, compared to the mean galectin-3 levels measured in the pigs that had no cardiac arrest. N = 3–11, *, p = 0.0036 10 days before cardiac arrest vs. 20 days before cardiac arrest in SCD pigs, †, p = 0.0002 10 days before cardiac arrest in SCD vs. survivors pigs. ( C , D ) shows representative immunofluorescence images of galectin-3-stained pig left ventricular sections with non-SCD and SCD. The bar diagram ( E ) in the lower panel shows a significantly increased level of galectin-3 in these animals. N = 5 per group. CTF, corrected total fluorescent intensity. Scale bar: 50 µm, magnification ×400.

    Article Snippet: For circulating gal3 measurement, we used a pre-coated gal3 (LSBio, Seattle, WA kit #LSF32161) specific 96 well strip microplates ELISA kit.

    Techniques: Comparison, Immunofluorescence, Staining

    Myocardial gene expression of galectin-3 after acute MI in mice. Panel A: Representative Trichrome-labeled section from infarct region. Panel B: Similarly stained section from remote region. The data columns represent fold-change of mRNA expression at different time-points of genomic profiling after acute MI induction. Panel C: Maximal galectin-3 expression was noted at 7-21 days with a chronic elevation present till 90 days post-MI. N = 3, each group; total, 8 time-points. * P < .05, 1-4 days vs 7-21 days; ** P < .05, 7-21 days vs 45-90 days. Panel D: The remote region showed minimal and statistically non-significant galectin-3 mRNA expression over time. MI indicates myocardial infarction.

    Journal: Biomarker Insights

    Article Title: The Therapeutic Potential of Blocking Galectin-3 Expression in Acute Myocardial Infarction and Mitigating Inflammation of Infarct Region: A Clinical Outcome-Based Translational Study

    doi: 10.1177/1177271918771969

    Figure Lengend Snippet: Myocardial gene expression of galectin-3 after acute MI in mice. Panel A: Representative Trichrome-labeled section from infarct region. Panel B: Similarly stained section from remote region. The data columns represent fold-change of mRNA expression at different time-points of genomic profiling after acute MI induction. Panel C: Maximal galectin-3 expression was noted at 7-21 days with a chronic elevation present till 90 days post-MI. N = 3, each group; total, 8 time-points. * P < .05, 1-4 days vs 7-21 days; ** P < .05, 7-21 days vs 45-90 days. Panel D: The remote region showed minimal and statistically non-significant galectin-3 mRNA expression over time. MI indicates myocardial infarction.

    Article Snippet: To measure serum galectin-3, a pre-coated galectin-3 specific 96-well strip microplate Enzyme-linked Immunosorbent Assays (ELISA) kit (R&D Systems; Minneapolis, MN, USA) was used, and optical densities were analyzed with standard galectin-3 concentrations (at ng/mL) based on the manufacturer’s recommendations.

    Techniques: Expressing, Labeling, Staining

    Post-MI myocardial galectin-3 expression and macrophage infiltration 1 week after LAD ligation. Wild-type mice showed scattered areas of galectin-3 positivity (Panel A1), which was absent in the myocardial sections obtained from galectin-3 KO mice (Panel A2). Panel A3 represents isotype control for galectin-3 antibody. Panel A4 represents quantitative analysis of Panels A1 and A2 (7 myocardial fields, each group). The macrophage-specific F4/80 staining in WT mice showed abundant positively stained cells in the infarct (red arrow) and peri-infarct (blue arrow) regions, especially in the areas of excess cardiomyocyte loss (Panel B1). The galectin-3 KO mice showed reduced F4/80 positive cellular infiltration compared with WT mice (Panel B2). Panel B3 represents isotype control for mouse F4/80 antibody. Panel B4 represents quantitative analysis of Panels B1 and B2 (12 myocardial fields, each group). KO indicates knock-out; LAD, left anterior descending; MI, myocardial infarction; WT, wild-type. * P < .05.

    Journal: Biomarker Insights

    Article Title: The Therapeutic Potential of Blocking Galectin-3 Expression in Acute Myocardial Infarction and Mitigating Inflammation of Infarct Region: A Clinical Outcome-Based Translational Study

    doi: 10.1177/1177271918771969

    Figure Lengend Snippet: Post-MI myocardial galectin-3 expression and macrophage infiltration 1 week after LAD ligation. Wild-type mice showed scattered areas of galectin-3 positivity (Panel A1), which was absent in the myocardial sections obtained from galectin-3 KO mice (Panel A2). Panel A3 represents isotype control for galectin-3 antibody. Panel A4 represents quantitative analysis of Panels A1 and A2 (7 myocardial fields, each group). The macrophage-specific F4/80 staining in WT mice showed abundant positively stained cells in the infarct (red arrow) and peri-infarct (blue arrow) regions, especially in the areas of excess cardiomyocyte loss (Panel B1). The galectin-3 KO mice showed reduced F4/80 positive cellular infiltration compared with WT mice (Panel B2). Panel B3 represents isotype control for mouse F4/80 antibody. Panel B4 represents quantitative analysis of Panels B1 and B2 (12 myocardial fields, each group). KO indicates knock-out; LAD, left anterior descending; MI, myocardial infarction; WT, wild-type. * P < .05.

    Article Snippet: To measure serum galectin-3, a pre-coated galectin-3 specific 96-well strip microplate Enzyme-linked Immunosorbent Assays (ELISA) kit (R&D Systems; Minneapolis, MN, USA) was used, and optical densities were analyzed with standard galectin-3 concentrations (at ng/mL) based on the manufacturer’s recommendations.

    Techniques: Expressing, Ligation, Staining, Knock-Out

    Comparison of serum galectin-3 level in healthy controls vs STEMI patients with either MACE-positive or MACE-negative outcome (Panel A) and in STEMI patients with reduced ejection fraction on ventriculogram (Panel B). Data are expressed as mean ± SEM. * P < .05 compared with control (Panel A); # P < .05 compared with MACE (Panel A); † P < .05 (Panel B). HF indicates heart failure; MACE, major adverse cardiovascular events defined as composite outcome of mortality, recurrent MI, stroke, and congestive HF hospitalization. MI, myocardial infarction; SEM, standard error of mean; STEMI, ST-elevation myocardial infarction.

    Journal: Biomarker Insights

    Article Title: The Therapeutic Potential of Blocking Galectin-3 Expression in Acute Myocardial Infarction and Mitigating Inflammation of Infarct Region: A Clinical Outcome-Based Translational Study

    doi: 10.1177/1177271918771969

    Figure Lengend Snippet: Comparison of serum galectin-3 level in healthy controls vs STEMI patients with either MACE-positive or MACE-negative outcome (Panel A) and in STEMI patients with reduced ejection fraction on ventriculogram (Panel B). Data are expressed as mean ± SEM. * P < .05 compared with control (Panel A); # P < .05 compared with MACE (Panel A); † P < .05 (Panel B). HF indicates heart failure; MACE, major adverse cardiovascular events defined as composite outcome of mortality, recurrent MI, stroke, and congestive HF hospitalization. MI, myocardial infarction; SEM, standard error of mean; STEMI, ST-elevation myocardial infarction.

    Article Snippet: To measure serum galectin-3, a pre-coated galectin-3 specific 96-well strip microplate Enzyme-linked Immunosorbent Assays (ELISA) kit (R&D Systems; Minneapolis, MN, USA) was used, and optical densities were analyzed with standard galectin-3 concentrations (at ng/mL) based on the manufacturer’s recommendations.

    Techniques: Comparison

    Comparison of early outcomes including length of hospitalization and early echocardiographic ( <72 hours) and angiographic (at time of PCI) parameters of myocardial dysfunction in STEMI patients in relation to galectin-3 level.

    Journal: Biomarker Insights

    Article Title: The Therapeutic Potential of Blocking Galectin-3 Expression in Acute Myocardial Infarction and Mitigating Inflammation of Infarct Region: A Clinical Outcome-Based Translational Study

    doi: 10.1177/1177271918771969

    Figure Lengend Snippet: Comparison of early outcomes including length of hospitalization and early echocardiographic ( <72 hours) and angiographic (at time of PCI) parameters of myocardial dysfunction in STEMI patients in relation to galectin-3 level.

    Article Snippet: To measure serum galectin-3, a pre-coated galectin-3 specific 96-well strip microplate Enzyme-linked Immunosorbent Assays (ELISA) kit (R&D Systems; Minneapolis, MN, USA) was used, and optical densities were analyzed with standard galectin-3 concentrations (at ng/mL) based on the manufacturer’s recommendations.

    Techniques: Comparison

    Demographic characteristics of STEMI patients and comparison between patients with and without MACE.

    Journal: Biomarker Insights

    Article Title: The Therapeutic Potential of Blocking Galectin-3 Expression in Acute Myocardial Infarction and Mitigating Inflammation of Infarct Region: A Clinical Outcome-Based Translational Study

    doi: 10.1177/1177271918771969

    Figure Lengend Snippet: Demographic characteristics of STEMI patients and comparison between patients with and without MACE.

    Article Snippet: To measure serum galectin-3, a pre-coated galectin-3 specific 96-well strip microplate Enzyme-linked Immunosorbent Assays (ELISA) kit (R&D Systems; Minneapolis, MN, USA) was used, and optical densities were analyzed with standard galectin-3 concentrations (at ng/mL) based on the manufacturer’s recommendations.

    Techniques: Comparison, Medications, Functional Assay

    Receiver-operating characteristic curve showing prediction of positive MACE outcome with galectin-3 compared with peak troponin and BNP. Area under the curve for galectin-3 is 0.914 (CI: 0.789-1.0; P < .05), BNP is 0.847 (CI: 0.709-0.985; P < .05), and peak troponin is 0.554 (CI: 0.258-0.851; P = .674). BNP indicates brain natriuretic peptide; CI, confidence interval; MACE, major adverse cardiovascular event.

    Journal: Biomarker Insights

    Article Title: The Therapeutic Potential of Blocking Galectin-3 Expression in Acute Myocardial Infarction and Mitigating Inflammation of Infarct Region: A Clinical Outcome-Based Translational Study

    doi: 10.1177/1177271918771969

    Figure Lengend Snippet: Receiver-operating characteristic curve showing prediction of positive MACE outcome with galectin-3 compared with peak troponin and BNP. Area under the curve for galectin-3 is 0.914 (CI: 0.789-1.0; P < .05), BNP is 0.847 (CI: 0.709-0.985; P < .05), and peak troponin is 0.554 (CI: 0.258-0.851; P = .674). BNP indicates brain natriuretic peptide; CI, confidence interval; MACE, major adverse cardiovascular event.

    Article Snippet: To measure serum galectin-3, a pre-coated galectin-3 specific 96-well strip microplate Enzyme-linked Immunosorbent Assays (ELISA) kit (R&D Systems; Minneapolis, MN, USA) was used, and optical densities were analyzed with standard galectin-3 concentrations (at ng/mL) based on the manufacturer’s recommendations.

    Techniques: